EMF Blocking plus EMF Shielding
Whether you live in an apartment or house or simply want to keep your home free of EMFs There are plenty of methods to reduce exposure. One of the easiest is to reduce your electronic device use. You could also consider EMF block paint to stop EMF radiation from reaching your house. Another method to protect your home against EMF radiations is to install an RF shielding canopy. This is a cloth of net that contains EMF shielding. It is utilized to prevent EMFs from entering a room. emf radiation blocker is to get your house equipped with an electrical enclosure. http://burnpipe3.xtgem.com/__xt_blog/__xtblog_entry/__xtblog_entry/33702285-information-about-emf-blocking-radiation?__xtblog_block_id=1#xt_blog are called Faraday cages.
A number of studies have proven studies have shown that nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation of cancer stem cells. This may account for the long-term responses seen in some patients with advanced HCC. However, the mechanism behind AM RF EMF's effect in cancer patients is not evident.
Aspects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumors were split into three groups. One group did not have exposure to RF EMF. Second group members were exposed to RF EMF at frequencies similar to the frequency used by humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumors was compared to that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism of cancer-specific AM RF EMF may be due to the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated by CACNA1H the protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications in the treatment and diagnosis of many cancers.
The tumours in the controls were never exposed to EMF from RF, and fed a standard mouse diet. The tumours in the HCCMF group were infected with Huh7 cells when they were five-seven weeks old. The tumors were removed in cases of excessive burden.
Click for more info from the three groups also displayed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, the tumours that were treated using RCF did not show any signs of shrinkage. The difference was substantial. The tumours treated with RCF showed necrosis, which is typical in tumours that are exposed to RCF. The possibility is that this necrosis is caused by an absence of oxygen in the more invasive tumours.
In sum, the results suggest that AM RF EMF is a powerful source of anticancer effects in vitro and in vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF produces these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the development of HCC tumours in vivo.