EMF Blocking in addition to EMF Shielding
Click here for more info if you live in an apartment or house or simply would like to ensure your home is free from EMFs, there are a number options to limit your exposure. One of the easiest is to limit your electronic device use. You can also turn to EMF block paint to prevent EMF radiation from reaching your house. Another way to shield your house against EMF radiation is to use an RF shielding canopy. It is a type of net that contains EMF shielding. It's used to block EMFs from entering a room. Another option is to get your house equipped with an electrical enclosure. They are referred to as Faraday cages.
A number of studies have proven how the non-ionizing energy of RF produces antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation of cancer stem cells. This may account for the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not yet clear.
Aspects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected to RF EMF at frequencies similar to the one used by humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumours was assessed against the effect of RCF. The results revealed that tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF could be based on the fact that cancer cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is caused by CACNA1H the protein that mediates tumour-specific Ca2+ influx. Click here! indicate that CACNA1H could have wider implications for the treatment and diagnosis of many cancers.
emf blocking clothing of the controls were never exposed EMF from RF, and fed a standard mouse diet. The tumors in HCCMF HCCMF group were treated with Huh7 cells after they were between five and seven weeks old. The tumors were removed in cases of excessive burden.
The tumours from the three groups also displayed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in the size of the tumour after 8 weeks. However, the tumors that were treated using RCF showed no shrinkage. The difference was substantial. The tumors treated by RCF had necrosis, which is typical when tumors are exposed to RCF. There is a possibility that the necrosis is caused by an absence of oxygen in the more invasive tumors.
In conclusion, the findings suggest that AM RF EMF has anticancer activity in vitro and in the vivo. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting influence on the growth of HCC tumours in living tissue.