EMF Blocking in addition to EMF Shielding
If you reside in a house or apartment, or you just need to ensure that your house is free of EMFs, there are a number of methods to reduce exposure. One of the most effective is to restrict the use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your home. Another way to shield your home from EMF radiation would be to install a RF shielding canopy. This is a cloth made of net which contains EMF shielding. It's used to block EMFs from entering a space. Another option is to have your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown studies have shown that nonionizing EMF has antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation in cancer-related stem cells. https://squareblogs.net/formknife2/emf-block-paint-in-addition-to-emf-shielding-s5g3 could explain the long-term responses seen in certain patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not evident.
Effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were split in three different groups. One group did not have exposure to RF EMF. Second group members were exposed RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumors was evaluated against that of RCF. The results revealed that cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. https://canvas.instructure.com/eportfolios/2026741/Home/EMF_Block_Paint_plus_EMF_Shielding in HCC cells is controlled by CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications for treatment and diagnosis of many cancers.
The tumours of the control group were not exposed to RF EMF, and were fed a normal diet of mice. The tumors of the HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups showed different growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, tumors which were treated by RCF didn't show shrinkage. The difference was highly significant. The tumours treated with RCF showed necrosis that is common in tumors that have been exposed to RCF. It is possible that the necrosis is caused by the lack of oxygen in the larger tumors.
In sum, the results indicate that AM RF EMF is a powerful source of anticancer effects in vitro and in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting effect on the development of HCC tumours in vivo.