EMF Blocking and EMF Shielding
If you reside in a house or apartment or just need to ensure that your house is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the most effective is to reduce your use of electronic devices. You can also turn to EMF block paint to stop EMF radiation from entering your house. Another method to protect your house from EMF radiation is to install a shielding canopy for RF. Additional resources is a fabric of net that contains EMF shielding. It's used to prevent EMFs from entering a room. Another option is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This may account for the long-term effects observed in some patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients is not clear.
Aspects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumours were divided in three different groups. One group did not have exposure RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to the one used by humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on the tumours was assessed against the effect of RCF. The results showed that the tumours treated with HCCMF had significant shrinkage. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The reason for cancer-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is controlled by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. block emf radiation suggest that CACNA1H may have broader implications in the treatment and diagnosis of different cancers.
The tumours of those in the group that were unaffected EMF from radiofrequency, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were infected with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed after they had a high burden.
The tumours from the three groups showed different growth curves. The HCCMF-treated tumors had a significant reduction in size of the tumor after 8 weeks. However, the tumors which were treated by RCF showed no signs of shrinkage. The difference was highly significant. The tumors treated with RCF had necrosis, which is common in tumours that are exposed to RCF. The possibility is that this necrosis is caused by the lack of oxygen in larger cancers.
In conclusion, the findings indicate that AM RF EMF has anticancer effects in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In block emf , AM RF EMF may have a long-lasting influence on the growth of HCC tumors in vivo.