EMF Block Paint plus EMF Shielding
Whether you live in a house or apartment or just would like to keep your home free of EMFs T here are plenty of methods to reduce exposure. One of the easiest is to limit the use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your house from EMF radiation is to put up an RF shielding canopy. It is a type of net that has EMF shielding and is used to stop EMFs from entering a room. Another option is to get your house equipped with a conductive enclosure. They are referred to as Faraday cages.
Several studies have shown how the EMF that is not ionized can cause anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. This could be the reason for the long-term responses seen in certain patients suffering from advanced HCC. However, the reason behind AM RF EMF's effect in cancer patients isn't evident.
Effects from AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumors were separated in three different groups. First, the group that was unaffected RF EMF. Another group of participants was subjected to RF EMF at frequencies similar to that used in humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on the tumours was compared to that of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism of tumor-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. block emf on HCC cells is controlled by CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have wider implications in the treatment and diagnosis of various cancers.
The tumours of the controls were never exposed EMF from RF, and fed a standard mouse diet. The tumors in those in the HCCMF group were infected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed in cases of excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, the tumours which were treated by RCF showed no shrinkage. The difference was highly significant. The tumours treated with RCF were able to show necrosis, which is typical in tumours that are exposed to RCF. There is block emf that this necrosis was caused by an absence of oxygen in the more invasive cancers.
In sum, the results show an AM-RF EMF is a powerful source of anticancer effects in vitro and in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. It is possible that AM RF EMF triggers these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the growth of HCC tumors in living tissue.