EMF Block Paint and even EMF Shielding
If you reside in a house or apartment or just need to ensure that your house is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most effective is to limit the use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from entering your house. Another method to protect your home against EMF radiations is to put up an RF shielding canopy. It's a kind of net that contains EMF shielding and is used to block EMFs from entering a room. Another option is to get your house equipped with an electrical enclosure. They are referred to as Faraday cages.
Numerous studies have demonstrated how the EMF that is not ionized can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. This may account for the long-term effects observed in patients with advanced HCC. However, blocking emf behind AM EMF's impact on cancer patients is not evident.
Aspects of AM RF EMFs on HCC tumour growth in vivo was studied in mice. The tumors were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to that of humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumors was evaluated against that of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, https://french-kock.hubstack.net/emf-blocking-plus-emf-shielding-1681432250 treated with RCF didn't show evidence of tumour shrinkage.
The mechanism behind cancer-specific AM RF EMF may be due to the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is caused by CACNA1H which is a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications in the diagnosis and treatment of various cancers.
The tumours in the control group were not exposed RF EMF, and were fed a normal mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells at the time they were five-seven weeks old. The tumors were removed when they showed excessive burden.
http://note.pearltrees.com/14006913/7d876d9a836b7a59c28433897b0d0e9c of the three groups showed distinct growth curves. The HCCMF-treated tumors had a significant reduction in tumour size after eight weeks. However, tumors which were treated by RCF did not show any shrinkage. The difference was highly significant. The tumours treated with RCF showed necrosis that is common in tumors that have been exposed to RCF. The possibility is that the necrosis was caused by the lack of oxygen in the larger tumours.
In sum, the results indicate an AM-RF EMF has anti-cancer properties in vitro as well as in live. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the development of HCC tumors in the vivo.