EMF Block Paint and EMF Shielding
No matter if you live in an apartment or house or just would like to keep your home free from EMFs There are plenty of ways you can reduce exposure. One of the most effective is to reduce your electronic device use. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another method to protect your house against EMF radiations is to install a RF shielding canopy. It's a kind of net that has EMF shielding and is used to block EMFs from entering a room. Another option is to get your home equipped with an electrical enclosure. They are referred to as Faraday cages.
A number of studies have proven studies have shown that the nonionizing EMF can cause anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This could explain the long-term responses seen in some patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients is not clear.
https://kold-appel.hubstack.net/emf-blocking-plus-emf-shielding-1681426348 on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumors were separated into 3 groups. The first group was not exposed to RF EMF. Another group of participants was subjected RF EMF at the same frequency to that used in humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is mediated through CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumors in those in the group that were unaffected EMF from RF, and fed a normal mouse diet. block emf in HCCMF HCCMF group were treated with Huh7 cells at the time they were five-seven weeks old. The tumors were then killed in cases of excessive burden.
The tumors in the three groups also showed distinct growth curves. The HCCMF-treated tumors had a significant reduction in size of the tumor after eight weeks. However, emf blocking clothing that were treated using RCF didn't show shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis, which is typical in tumours that are exposed to RCF. It is possible that this necrosis was due to the lack of oxygen in the larger tumors.
In sum, the results show that AM RF EMF has anti-cancer properties in vitro as well as in vivo. A number of studies have proven it is true that AM RF EMF produces measurable tumour shrinkage for HCC patients. The possibility is that the AM EMF causes these effects because of CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in living tissue.